the strange reality of strict calvinism is that it makes a mockery of God's claims to hate, abhort, or even be theoretically opposed to evil
Because we all know that God's response to the Flood was, "I REALLY liked that, it was AWESOME, it satisfied my will and purposes to obliterate my creation!"
1) If God knew how things would go, exercising his sovereignty in that way is only a little different
2) Evil begins BEFORE man's fall! Satan fell first! We know little about this, but locating first-evil in *human* free will is an error.
The key point is simply that the origin of evil is certainly *before* "original sin."
This is formally equivalent to the question, "Could the Father discontinue the existence of the Son?" since both are questions about if God has sovereignty over His own attributes.
Who can say! Not even the Son was told the day or the hour! But if God is taking His time there must be a reason. We can't reason about what that may be, as God has not revealed it.
.... love me all my Presby family and friends (literally both sides of the family and many of my best friends!)....
.... and your theology is interesting...
.... but very, very, very wrong.
More from Lyman Stone 石來民
Hogan Gidley: Trump is "the most masculine person to ever hold the White House as the president of the United States" https://t.co/fcoYWyaEhz
— Eliza Relman (@eliza_relman) January 11, 2021
Or Teddy Roosevelt. Or Dwight Eisenhower. Or Andrew Jackson. Or Abraham Lincoln. Or George Washington. Or Zachary Taylor. Or any of numerous presidents who were honest-to-goodness battle-hardened warriors.
James Monroe fought the Hessians at Trenton and nearly died of wounds sustained there, then wintered in Valley Forge, then fought until Monmouth, then repeatedly tried to raise new regiments for the war until he went bankrupt doing it.
James Monroe, of the Era of Good Feelings, longest serving president of all time.... was in the boats crossing the icy Delaware.
Andrew Jackson was in a duel. He was shot in the chest right by his heart.
But he didn't go down. He stood there and, while bleeding out, steadily took aim and killed the dude who shot him.
Stone cold.
So first off, at this point the evidence is pretty clear that SSRIs and other anti-anxiety/anti-depression drugs truly don't do very much. Their average effects are beneath clinical significance, as I tweeted about here:
What's the best recent empirical assessment of SSRI/SNRI effectiveness which deals with heterogeneity and long-term effects in a plausible way?
— Lyman Stone \u77f3\u4f86\u6c11 (@lymanstoneky) December 4, 2020
Basically, the problem these drugs face is that while they actually see relatively LARGE effects.... but that placebos in those trials ALSO see large effects (and most untreated depression improves within a year anyways).
So basically you have this problem where:
1. The condition tends to improve on its own in a majority of cases
2. Placebo effects for the condition are unusually large
Which means the large crude effects of SSRIs get swamped.
So that raises two new questions.
1. (Not my focus here) Are we treating these conditions appropriately given their untreated prognosis is usually (though certainly not always!!) "goes away in a few months"?
2. Why are placebo effects so unusually large?
More from For later read
We want to collate all references to DRASTIC in academic papers & media articles
Here are a few:
medium article by @emmecola
thorough report by @netpoette
@ColinDavdButler 's Paper
Please add any links to this thread. Tks!
2. More References
Papers by @MonaRahalkar and @BahulikarRahul
Papers by @Rossana38510044 and @ydeigin
Medium articles & papers by
@gdemaneuf & @Rdemaistre
Papers by @flavinkins (Daoyu Zhang)
Papers by "Anon" & "interneperson"
French News - le Monde
Can anyone remember any more?
3. More References
Papers & Blog Posts by @Harvard2H (Sirotkin & Sirotkin)
260 Questions for WHO collated by @billybostickson
If you find mentions of our individual names or "DRASTIC" in Papers or News, please forward here to this thread as links or screenshots.
Histoire du COVID-19 – chapitre 6 - Partie 2 : Pourquoi le séquençage complet du virus RaTG13 n'a pas été communiqué par Shi Zheng Li avant février 2020 ? https://t.co/MYEZZSAzaE
SARS-CoV-2: lab-origin hypothesis gains traction
BY ANNETTE GARTLAND ON OCTOBER 12, 2020
https://t.co/sPs1y8Herg
https://t.co/w7koHyMJjL
The article mentions a “10 nucleotide linker” (GCAUAUGACU) in the poly-A tail. This is described in the patent link below (Modification of RNA, producing an increased transcript stability and translation
Here is a link to the full mRNA code if you wish to download it, blast it or make up a batch in your garage
The mRNA sequences used for Moderna mRNA-1273 & Pfizer BNT162b2 mRNA vaccines for COVID-19 (Direct link in Word Format). WHO International Nonproprietary Name Program # 11889 "Messenger RNA encoding the full-length SARS-CoV-2 spike glycoprotein"https://t.co/zTb7B0Apic pic.twitter.com/8tZxAZWI5S
— Roland Baker (@RolandBakerIII) December 24, 2020
An overview of the encoded spike
Moderna's mRNA-1273 & Pfizer's BNT162b2 consist of mRNA 3821 nucleotides long encoding *all* 1273 amino acids of the Spike including a 2 Proline-stabilized RBD and this includes the NTD (blue in monomer, dark gray in trimer attached to antibodies). AA 64, 66, 187, 213, 214 red. pic.twitter.com/4MX1ByAsrR
— Roland Baker (@RolandBakerIII) December 19, 2020
Initial mouse
News: NIH-Moderna investigational COVID-19 vaccine shows promise in mouse studies https://t.co/7JYuUyZT45
— NIH (@NIH) August 5, 2020