https://t.co/eAmtY241ug
They are now saying my tweets are misleading. It's another lie. They are the ones who are misleading, and I will now prove it in this thread. They are lying about the safety of the vaccines:
https://t.co/eAmtY241ug
More from Robin Monotti MA FRSA ⭐
Italian researchers: vaccines will not work against SARSCoV2 because this virus does not only replicate in human cells like other viruses, this one replicates through bacteria too. This is the fundamental reason why antibiotics work & vaccines will not:
Here is the Italian-EU scientific study indicating SARSCoV2 replicates in bacteria, not only human cells, and that is why antibiotics work and these vaccines will not:
"The preliminary results suggest that SARS-CoV-2 replicates in bacterial
2 of the 4 authors of the study work at the European Commission. Another works at an Italian medical research facility called Craniomed: https://t.co/EETSM3nb3T
You can find all of CRANIOMED's Carlo Brogna's published scientific research articles here, take a look:
Here is the Italian-EU scientific study indicating SARSCoV2 replicates in bacteria, not only human cells, and that is why antibiotics work and these vaccines will not:
"The preliminary results suggest that SARS-CoV-2 replicates in bacterial
2 of the 4 authors of the study work at the European Commission. Another works at an Italian medical research facility called Craniomed: https://t.co/EETSM3nb3T
You can find all of CRANIOMED's Carlo Brogna's published scientific research articles here, take a look:
The problem with meta-analysis like this is that it obfuscates the most important issue of treatment, which is timing.
This meta-analysis of controlled trials only looks at hospitalized patients. How long were the patients ill for before being hospitalized? One week? Two? Three? Too late for zinc ionophores (HCQ) (+ZINC? No zinc no point..) to work. Severe illness becomes bacterial in nature.
Was azythromycin administered when the bacterial infections were also too advanced? I have seen Azythromycin work with my very own eyes but that's not to say that if administered too late it may not save the patient. How many patients were given AZT & ventilated? It's all timing.
All the meta-analysis is telling us is if you leave it too late you may have missed the early window for antiviral zinc treatment (Zn+HCQ) & that if you are given AZT when you are ventilated or very severe it may too late for it to save you & corticosteroids may be last resort.
And of course antibiotics need also probiotics, or they may harm the bacterial flora which is part of the immune response. Difficult to tell from a meta-analysis how this problem was managed.
#BMJResearch update: Corticosteroids probably reduce mortality and mechanical ventilation in patients with covid-19 compared with standard care, whereas azithromycin, hydroxychloroquine, interferon-beta, and tocilizumab may not reduce either https://t.co/oQ3lTWUqaz
— The BMJ (@bmj_latest) December 18, 2020
This meta-analysis of controlled trials only looks at hospitalized patients. How long were the patients ill for before being hospitalized? One week? Two? Three? Too late for zinc ionophores (HCQ) (+ZINC? No zinc no point..) to work. Severe illness becomes bacterial in nature.
Was azythromycin administered when the bacterial infections were also too advanced? I have seen Azythromycin work with my very own eyes but that's not to say that if administered too late it may not save the patient. How many patients were given AZT & ventilated? It's all timing.
All the meta-analysis is telling us is if you leave it too late you may have missed the early window for antiviral zinc treatment (Zn+HCQ) & that if you are given AZT when you are ventilated or very severe it may too late for it to save you & corticosteroids may be last resort.
And of course antibiotics need also probiotics, or they may harm the bacterial flora which is part of the immune response. Difficult to tell from a meta-analysis how this problem was managed.
I have now re-examined this document:
It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"
"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."
"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"
"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."
On the 19th March 2020 the WHO released this guidance intended for healthcare workers (HCWs), healthcare managers and IPC teams at the facility level & at national and district/provincial level:https://t.co/C4aV2BnMPj pic.twitter.com/tCk1EyLskV
— Robin Monotti (@robinmonotti) December 21, 2020
It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"
"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."
"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"
"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."
More from Category c19
Let's talk about MASKS!
Thread 1:
Masks increase mortality because breathing through them nebulizes aerosols into smaller ones which bypass mucosal immunity & reach all the way into the alveoli, leading to acute respiratory distress syndrome (ARDS).
"Aerosols..within the most breathable size range between 0.5 & 5 μm, can carry SARS-CoV-2 deep to the terminal alveoli..if this transmission pathway does exist, it would bypass the mucociliary clearance & incubation period of the virus in the upper
The filtration material itself of N95's average pore size ~0.3−0.5 μm does not block finer aerosol laden with virions penetration, not to mention surgical masks.
Thread 1:
If you study the field you will see viral nebulization is a technique used to get higher infectivity of viruses. This is used with ventilator patients suffering from pseudomonas aeruginosa infections. Nebulize bacteriaphage to get deep into the lungs and kill bacteria. pic.twitter.com/82lNRMrXl4
— Kevin McKernan \U0001f642 (@Kevin_McKernan) October 24, 2020
Masks increase mortality because breathing through them nebulizes aerosols into smaller ones which bypass mucosal immunity & reach all the way into the alveoli, leading to acute respiratory distress syndrome (ARDS).
"Aerosols..within the most breathable size range between 0.5 & 5 μm, can carry SARS-CoV-2 deep to the terminal alveoli..if this transmission pathway does exist, it would bypass the mucociliary clearance & incubation period of the virus in the upper
The filtration material itself of N95's average pore size ~0.3−0.5 μm does not block finer aerosol laden with virions penetration, not to mention surgical masks.
1/ Thread – THE YEAR OF THE RAT
“15 days to slow the spread” began one year ago. It was March 2020, the Year of the Rat. Never was any year more fitting of the name. Never in history have the people been betrayed so callously, flagrantly and absolutely by their alleged leaders.
2/ They were betrayed by their WHO
3/ and their national health
4/ They were betrayed by medical
5/ and their peer
“15 days to slow the spread” began one year ago. It was March 2020, the Year of the Rat. Never was any year more fitting of the name. Never in history have the people been betrayed so callously, flagrantly and absolutely by their alleged leaders.
2/ They were betrayed by their WHO
3/ and their national health
4/ They were betrayed by medical
Despite ignorance by many in the West, this article by The Lancet is a powerful endorsement of China\u2019s successful pandemic response. Hate to read stories by those paparazzi journalists who are experts at spinning but have little knowledge of science. https://t.co/Q8rKwwTPsI pic.twitter.com/436BEmx9nl
— Chen Weihua \uff08\u9648\u536b\u534e\uff09 (@chenweihua) October 16, 2020
5/ and their peer
6/ Eurosurveillance, a journal whose editors coincidentally includes Mr. Drosten, takes only a single day for the peer review process and publishes the hastily compiled PCR protocol on January 22. https://t.co/cWX3UdKeID
— Pace \U0001f642 (@theotherphilipp) February 25, 2021
1) THERE IS A VERY REAL, CREDIBLE AND SIGNIFICANT DANGER THAT mRNA SPIKE PROTEIN THERAPIES WILL INDUCE PRION OR PRION-LIKE DISEASE
A paper from 2016 shows that prion and prion-like proteins can also spread and propagate from cell to cell through macropinocytosis. It is also
2) noted that the Spike Protein of SARS-CoV-2 can enter cells through macropinocytosis, without the presence of the entire virion. Given the recent Tweet to me by @endocdoc, where he recently saw a patient develop Creutzfeld-Jakob after COVID-19, the extant paper showing CJD
3) CJD after COVID-19 infection and the recent death of the head of Astra-Zeneca research from CJD should be concerning to all. Especially if he gave himself the AZ therapy during Stage 1 or Stage 2 trials. This would give a possible timeframe for onset.
Again, all Spike Protein
4) mRNA and DNA should be paused until long term animal studies can be
5)
https://t.co/PWOgyPoG5d
A paper from 2016 shows that prion and prion-like proteins can also spread and propagate from cell to cell through macropinocytosis. It is also
2) noted that the Spike Protein of SARS-CoV-2 can enter cells through macropinocytosis, without the presence of the entire virion. Given the recent Tweet to me by @endocdoc, where he recently saw a patient develop Creutzfeld-Jakob after COVID-19, the extant paper showing CJD
3) CJD after COVID-19 infection and the recent death of the head of Astra-Zeneca research from CJD should be concerning to all. Especially if he gave himself the AZ therapy during Stage 1 or Stage 2 trials. This would give a possible timeframe for onset.
Again, all Spike Protein
4) mRNA and DNA should be paused until long term animal studies can be
5)
https://t.co/PWOgyPoG5d
