BC LOCKDOWNS
Saved by @trestrange

I’m not just a lockdown sceptic. I know lockdowns increase infections rather than decrease them because I read research first hand. I’m a citizen in a democracy who wants Government policy to be evidence-based, not epidemiological modelling based. Crazy, I know.

 Lockdowns increase infections because they lower immunity & therefore increase the disease. After hospitals & care homes households account for the largest number of transmissions. Schools/universities act as a break in transmission of the disease. All evidence based.
Transmission does not mean infection. I can transmit SARSCoV2 but I can't transmit Covid19. Whether SARSCoV2 develops at all into mild or severe Covid19 depends entirely on the immune system of the recipient. Early treatment prevents severe Covid19, the right treatment cures it.
"Children act more as a brake on infection," said Prof. Reinhard Berner, the head of pediatric medicine at Dresden University Hospital and leader of the study. "Not every infection that reaches them is passed on."
https://t.co/FokkJhgtzM
Closing schools increases transmission of respiratory viral infection because children act as a break on the community transmission of the virus. We have known this since 1918:
https://t.co/TPRYQ1LAAJ
"We confirm that adding school and university closures to case isolation, household quarantine, and social distancing of over 70s would lead to more deaths compared with the equivalent scenario without the closures of schools and universities."
https://t.co/99FR4IqbJj
"Stringency of the measures settled to fight pandemia, including lockdown, did not appear to be linked with death rate."
https://t.co/5N06jgA0Bg
"We explored two MODELS DEVELOPED BY IMPERIAL COLLEGE..Inferences on effects of Non Pharmaceutical Interventions are non-robust and highly sensitive to model specification. CLAIMED BENEFITS OF LOCKDOWN APPEAR GROSSLY EXAGGERATED."
https://t.co/gX6xuQz5EY
There is no correlation whatsoever between mortality rates & lockdowns. There are instead distinct correlations between mortality rates & vitamin D levels:
"Correlations have been shown between the historic prevalence of vitamin D deficiency and COVID-19 mortality per million by country. This has been shown for European countries"
https://t.co/0FwTjxpKtR
There are also noticeable correlations between Omega-3 levels & low mortality rates:
"Global survey of the omega-3 fatty acids, docosahexaenoic acid and eicosapentaenoic acid in the blood stream of healthy adults"
Green=High Omega-3 levels
Red=Low Omega-3 levels
https://t.co/h0HzV1Y5gN
Why vitamin D rather than lockdowns reduces mortality:
"Vitamin D deficiency enhances the cytokine storm, thereby, it is a risk factor for and/or a driver of the excessive and persistent inflammation, which is a main characteristic of ARDS and may be considerably lethal in subjects with SARS‐CoV‐2 infection"
https://t.co/vAw4pphsSC
"When a T cell is exposed to a foreign pathogen, it extends a signalling device or 'antenna' known as a vitamin D receptor, with which it searches for vitamin D,", and if there is an inadequate vitamin D level, "they won't even begin to mobilize." 
https://t.co/RPagpdYkll
Why Omega 3s reduce mortality. Thread: https://t.co/UuZjlc5Bm1
"The most restrictive non‐pharmaceutical interventions (NPIs) for controlling the spread of COVID‐19 are mandatory stay‐at‐home and business closures..we do not find significant benefits on case growth of more restrictive NPIs."
https://t.co/2STxtMHVmA

More from Robin Monotti

Robin Monotti FRSA MA BSc
Robin Monotti FRSA MA ...
@robinmonotti
BAYES' THEOREM: The basic reason we get so many false positives to COVID19. The disease is so rare that the number of false positives greatly outnumbers the people who truly have the disease: THE MATHS:
https://t.co/oLHyxYJW9H


"Suppose that you are worried that you might have a rare disease. You decide to get tested, and suppose that the testing methods for this disease are correct 99 percent of the time"

"Suppose this disease is actually quite rare, occurring randomly in the general population in only one of every 10,000 people. If your test results come back POSITIVE, what are your chances that you actually have the disease? LESS THAN 1% chance that you have the disease!"


"The basic reason we get such a surprising result is because the disease is so rare that the number of false positives greatly outnumbers the people who truly have the disease"


Say mass testing of the contagious virus was done to 1 million people. In that million, 100 will really have the disease, 99 will be correctly diagnosed as having it. 999,900 of the million will not have the disease, but of those about 9,999 will be false positives! #BayesTheorem
CATEGORY C19
Robin Monotti
Robin Monotti
@robinmonotti
I have now re-examined this document:


It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"

"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."

"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"

"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."
HEALTH
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Robin Monotti FRSA \u2b50
Robin Monotti FRSA ⭐...
@robinmonotti
Italian researchers: vaccines will not work against SARSCoV2 because this virus does not only replicate in human cells like other viruses, this one replicates through bacteria too. This is the fundamental reason why antibiotics work & vaccines will not:

Here is the Italian-EU scientific study indicating SARSCoV2 replicates in bacteria, not only human cells, and that is why antibiotics work and these vaccines will not:

"The preliminary results suggest that SARS-CoV-2 replicates in bacterial

2 of the 4 authors of the study work at the European Commission. Another works at an Italian medical research facility called Craniomed: https://t.co/EETSM3nb3T


You can find all of CRANIOMED's Carlo Brogna's published scientific research articles here, take a look:
CATEGORY C19
Robin Monotti
Robin Monotti
@robinmonotti
The problem with meta-analysis like this is that it obfuscates the most important issue of treatment, which is timing.


This meta-analysis of controlled trials only looks at hospitalized patients. How long were the patients ill for before being hospitalized? One week? Two? Three? Too late for zinc ionophores (HCQ) (+ZINC? No zinc no point..) to work. Severe illness becomes bacterial in nature.

Was azythromycin administered when the bacterial infections were also too advanced? I have seen Azythromycin work with my very own eyes but that's not to say that if administered too late it may not save the patient. How many patients were given AZT & ventilated? It's all timing.

All the meta-analysis is telling us is if you leave it too late you may have missed the early window for antiviral zinc treatment (Zn+HCQ) & that if you are given AZT when you are ventilated or very severe it may too late for it to save you & corticosteroids may be last resort.

And of course antibiotics need also probiotics, or they may harm the bacterial flora which is part of the immune response. Difficult to tell from a meta-analysis how this problem was managed.
MOVIE
Robin Monotti Graziadei
Robin Monotti Graziade...
@robinmonotti
"PCR is just a process..it does not tell you that you are sick."

Kary Mullis Inventor of the PCR test & Nobel Prize in
PCR

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#DoYouKnow -The place where
Lalitha Sahasranamam -1000 names of Devi originated.

It is in Lalithambigai temple -Thirumeeyachur,Near Mayiladuthurai,TN

There is a Legend #story & it is related to this beautiful #sculpture of Kshetrapuraneswarar & Ambal Lalithambigai #Thread 👇


#Legend #story
It is believed that this is birth place of Kashyapa Maharishi’s sons, Arunan& Garudan. Arunan(charioteer of Surya)was physically handicapped.He was a Shiva devotee.Arunan wanted to visit Kailash to have Shiva Darshan,but Surya Dev denied permission.

So Arunan disguised as a girl & was on his way to Kailash.On the way Surya Dev saw this girl & outraged her modesty.Shiva was very angry with Suryan for misbehaviour & cursed him to get darkened. Whole world plunged into darkness.Surya Dev pleaded to Bhagwan Shiv to forgive him.

Shiva Bhagwan asked him to go to Thirumiyachur & worship him there. As advised, Surya Dev performed rigorous penance, placed murthi of Shiva& Parvathy on an elephant & took them to the clouds.Shiva in this temple is known as Meganathaswamy,Devi is known as Lalithambigai


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Sarvaripati Shiva is one of the most fearsome manifestations of Mahadev. Assigned with destroying all of the universes at the end of time, Shiva is one of the most fearsome warriors. He is the Mrityunjaya.
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TRISHULA: the 3 pointed blades denote creation, maintenance, and destruction or the 3 Kalas – Past, Present and Future. They also represent 3 Gunas – Sattva, Rajas and Tamas).
Shiva’s trishul can only be stopped by Shiva himself.
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PARASHU: axe with 4 cutting edges which Shiva gave to Parashurama. In his anger, Parashurama used the parashu to get rid of the all the Earth’s tyrannical kshatriya caste twenty-one times over.
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CHANDRAHASA SWORD: The divine sword Chandrahasa literally means ‘the laughter of the moon’. Ravana gave this sword to Indrajit on different occasions especially to fight against Devas.
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They share the same Japanese Voice actress! Rie Kugimiya.
Usually this is a stretch reason, but considering everything else, it’s not unlikely that it’s the same character.

Secondly, their Appearances.
The both have very long white hair with identical bang style, and a very noticeable small piece of hair on top that curls upwards. That’s a defining part Of all of Kiana’s hairstyles, and for it to be shared with this god says something.


Another notable thing is their eyes. They both have Glowing golden eyes with very similar appearances. They contain a ring, and a 4-point star pupil. Resemblant of a scope if you will. That’s The Herrscher of Void’s defining characteristic


If you’d even like to take a look at their outfits, there’s resemblances including the black fur collar, the designs on the torso and chest, and the use of something similar to coat tails/skirt. The Herrscher can change their outfit and they aren’t stuck with one. Sirin does this
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