BC CATEGORY C19

Robin Monotti FRSA ⭐
@robinmonotti 3 years, 1 month ago 510 views

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"Men with higher levels of 1,25(OH)2D and higher activation ratios, but not 25(OH)D itself, are more likely to possess butyrate producing bacteria that are associated with better gut microbial health."

"Several studies suggest that gut microbiota alter intestinal vitamin D metabolism (VDM), and probiotic supplements can affect circulating vitamin D levels."

"Because the serum 25(OH)D correlates with overall vitamin D storage, it is the preferred clinical measure to assess vitamin D sufficiency."

"Clinically, serum 25(OH)D levels ≥20 ng/ml are considered adequate while 25(OH)D levels <20 ng/ml are defined as vitamin D deficiency."

"However, it is the active form of vitamin D, 1,25(OH)2D, that interacts specifically with the vitamin D receptor (VDR) and transacts gene expression."

"Those men with the highest compared to lowest 1,25(OH)2D and activation ratios are more likely to possess butyrate-producing bacteria that are associated with favorable gut microbial health."

"25(OH)D levels vary with site and sun exposure, but 1,25(OH)2D levels do not follow this association."

"the serum 1,25(OH)2D was the factor that explained the highest proportion of the variance in α-diversity (e.g., bacterial species diversity within an individual) at just over 5%"

"Greater α-diversity [gut bacterial species diversity] is associated with higher 1,25(OH)2D levels and larger vitamin D activation and catabolism ratios."

"Overall, apart from known correlates such as race and geographic locations, measures of vitamin D metabolic flux were remarkably associated with microbial β-diversity"

"We report robust correlations between the vitamin D metabolites, 1,25(OH)2D and 24,25(OH)2D, and the gut microbiome in 567 older men representing six geographic sites across the United States"

"Those men with higher levels of 1,25(OH)2D had greater α-diversity, even after adjusting for previously characterized determinants of microbial diversity including age, geographical origin, race, PPI, and antibiotic use."

"Serum 25(OH)D is the preferred clinical measure because it is representative of overall body stores of vit D; however, our results suggest it is the regulation of VDM, reflected by active hormone & metabolic ratios rather than body stores that have the most health implications."

"The positive association between diversity metrics and vitamin D activation and catabolism ratios suggests that physiologically normal vitamin D flux is more likely to occur in individuals with healthy microbiomes."

"In summary, we provide strong evidence of important interactions between host vitamin D signaling and the health of the gut microbiome in older men."

More from Robin Monotti FRSA ⭐

Robin Monotti
@robinmonotti

The problem with meta-analysis like this is that it obfuscates the most important issue of treatment, which is timing.

#BMJResearch update: Corticosteroids probably reduce mortality and mechanical ventilation in patients with covid-19 compared with standard care, whereas azithromycin, hydroxychloroquine, interferon-beta, and tocilizumab may not reduce either https://t.co/oQ3lTWUqaz
— The BMJ (@bmj_latest) December 18, 2020

This meta-analysis of controlled trials only looks at hospitalized patients. How long were the patients ill for before being hospitalized? One week? Two? Three? Too late for zinc ionophores (HCQ) (+ZINC? No zinc no point..) to work. Severe illness becomes bacterial in nature.

Was azythromycin administered when the bacterial infections were also too advanced? I have seen Azythromycin work with my very own eyes but that's not to say that if administered too late it may not save the patient. How many patients were given AZT & ventilated? It's all timing.

All the meta-analysis is telling us is if you leave it too late you may have missed the early window for antiviral zinc treatment (Zn+HCQ) & that if you are given AZT when you are ventilated or very severe it may too late for it to save you & corticosteroids may be last resort.

And of course antibiotics need also probiotics, or they may harm the bacterial flora which is part of the immune response. Difficult to tell from a meta-analysis how this problem was managed.

Robin Monotti
@robinmonotti

I’m not just a lockdown sceptic. I know lockdowns increase infections rather than decrease them because I read research first hand. I’m a citizen in a democracy who wants Government policy to be evidence-based, not epidemiological modelling based. Crazy, I know.

Lockdowns increase infections because they lower immunity & therefore increase the disease. After hospitals & care homes households account for the largest number of transmissions. Schools/universities act as a break in transmission of the disease. All evidence based.

Transmission does not mean infection. I can transmit SARSCoV2 but I can't transmit Covid19. Whether SARSCoV2 develops at all into mild or severe Covid19 depends entirely on the immune system of the recipient. Early treatment prevents severe Covid19, the right treatment cures it.

"Children act more as a brake on infection," said Prof. Reinhard Berner, the head of pediatric medicine at Dresden University Hospital and leader of the study. "Not every infection that reaches them is passed

Closing schools increases transmission of respiratory viral infection because children act as a break on the community transmission of the virus. We have known this since 1918:
https://t.co/TPRYQ1LAAJ

Robin Monotti
@robinmonotti

I have now re-examined this document:

On the 19th March 2020 the WHO released this guidance intended for healthcare workers (HCWs), healthcare managers and IPC teams at the facility level & at national and district/provincial level:https://t.co/C4aV2BnMPj pic.twitter.com/tCk1EyLskV
— Robin Monotti (@robinmonotti) December 21, 2020

It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"

"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."

"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"

"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."

Robin Monotti Graziade...
@robinmonotti

LIST OF QUESTIONS FOR MPS
From @ClareCraigPath and Dr Jonathan Engler:
.
1.Why are SARS-CoV-2 antibody levels flat or dropping across all age groups since May if the pandemic is still going?

2.What percentage of the population is assumed to have had prior immunity to SARS-CoV-2 in the SAGE forecasting models?

3.Why do 50% of household members not catch SARS-CoV-2 from infected persons with whom they live?

4.Why have Japan and South Korea not had any serious outbreak if the human species has no prior immunity to SARS-CoV-2?

5.What percentage of the population of the UK is assumed to be immune to COVID-19 (including prior immunity) as of this date?

Robin Monotti FRSA
@robinmonotti

I have studied the pandemic response of many countries. One of them got a key element right, which resulted in very few deaths: Japan. Their response was to keep people OUT of hospitals. A policy of early home treatment works best, hospitals only for severe cases & NO INTUBATIONS

JAPAN: "No restrictions were placed on residents’ movements, and businesses from restaurants to hairdressers stayed open. No high-tech apps that tracked people’s movements were deployed." RESULT: Among the lowest deaths in the

Saving the NHS simply means providing early treatment kits in pharmacies & avoiding hospitals for Covid19 patients. For Covid19 infections during a pandemic, hospitals are the problem, not the solution.

I have read accounts of people with high fever literally turned away from hospitals, yet being told by phone what treatment protocol to follow. That was the best pandemic response by far, NOT lockdowns.

JAPAN had some of the lowest Covid19 mortality in the world:
-NO LOCKDOWN
-NO TEST AND TRACE
-NO BUSINESS CLOSURES
-NO HOSPITAL FOR NON CRITICAL CASES
-HOME TREATMENT
-HEALTHY DIET
-LOW OBESITY

More from Category c19

$AL \U0001f642$

AL 🙂
@Metabo_PhD

Last week, Dr. Theresa Tam @CPHO_Canada & the Ontario Scientology table released their latest model that garnered criticism, ridicule, & shame at an international level. The now famous Covid Erection had risen 🍆📈

#Canada #Ontario #cdnpoli #onpoli #COVID19

The Covid Erection model predicted ~5000 cases by the end of February. Well, now that we’re at February’s end, Ontario recorded ~1250 cases. It would seem that the Covid Erection encountered some...ahem, erectile dysfunction 📉

#Canada #Ontario #cdnpoli #onpoli

Since the Covid Erection model had become flaccid, Dr. Tam @CPHO_Canada needed another story to continue the propaganda of fear; public health by dread & distress. So she came out today to say that Canada was experiencing 75X more cases than the spring peak

#Canada #cdnpoli

The spring peak in Canada had ~2000 cases. Today, Canada reported ~3000 cases. That would be 1.5X more cases, not 75X. Dr. Tam @CPHO_Canada just inflated the case counts by only 50X, or what we call here in Canada, by a Fisman

#Canada #cdnpoli #Ontario #onpoli #COVID19

Canada (& Ontario’s) public health officials & politicians have thoroughly embarrassed the discipline of public health & made mockery of the field of medicine. From the MDs fake crying in the news to the high school teachers pretending to be biostatisticians. We’ve become a joke.

$Robin Monotti FRSA \u2b50$

Robin Monotti FRSA ⭐...
@robinmonotti

Italian researchers: vaccines will not work against SARSCoV2 because this virus does not only replicate in human cells like other viruses, this one replicates through bacteria too. This is the fundamental reason why antibiotics work & vaccines will not:

Here is the Italian-EU scientific study indicating SARSCoV2 replicates in bacteria, not only human cells, and that is why antibiotics work and these vaccines will not:

"The preliminary results suggest that SARS-CoV-2 replicates in bacterial

2 of the 4 authors of the study work at the European Commission. Another works at an Italian medical research facility called Craniomed: https://t.co/EETSM3nb3T

You can find all of CRANIOMED's Carlo Brogna's published scientific research articles here, take a look:

Alex Berenson
@AlexBerenson

1/ An excellent new paper from Denmark that tracks every vaccination given to nursing home residents there shows an 40% RISE in infections immediately after the first @pfizer dose and no efficacy at any point. Protection in this population after “full” vaccination is 64%...

2/ The researchers also tracked health-care workers, the other group in Denmark to receive the vaccine first. They actually had an even LARGER rise in infections immediately following dose 1, but overall higher protection at “full” vaccination (a week after dose 2) - 90%...

3/ This paper is the best yet (because it’s the least political). It is also in line with the Israeli and English data show once you wade through their spin. The takeaways are: The vaccine is much less effective in the people who need it most and DON’T DELAY THE SECOND SHOT...

4/ By the way, i#depending on how long the vaccine provides protection, its OVERALL effectiveness will be below whatever the peak figure is, maybe well below - you need a lot of good weeks to make up for that bolus at the beginning - but I don’t even want to go there.

5/ This paper also shows why the raw Israeli numbers are trash - if infections are declining overall (as they were in Israel and Denmark in February) looking at infections in the vaccinated population without adjusting for that trend markedly overestimates the vaccine effect.

Dr. Thomas Binder, MD
@Thomas_Binder

1/12

RT-PCR corona (test) scam

Symptomatic people are tested for one and only one respiratory virus. This means that other acute respiratory infections are reclassified as

4/10

...indication, first of all that testing for a (single) respiratory virus is done outside of surveillance systems or need for specific therapy, but even so the lack of consideration of Ct, symptoms and clinical findings when interpreting its result. https://t.co/gHH6kwRdZG
— Dr. Thomas Binder, MD (@Thomas_Binder) October 22, 2020

2/12

It is tested exquisitely with a hypersensitive non-specific RT-PCR test / Ct >35 (>30 is nonsense, >35 is madness), without considering Ct and clinical context. This means that more acute respiratory infections are reclassified as

6/10

The neither validated nor standardised hypersensitive RT-PCR test / Ct 35-45 for SARS-CoV-2 is abused to mislabel (also) other diseases, especially influenza, as COVID-19.https://t.co/AkFIfTCTkS
— Dr. Thomas Binder, MD (@Thomas_Binder) October 22, 2020

3/12

The Drosten RT-PCR test is fabricated in a way that each country and laboratory perform it differently at too high Ct and that the high rate of false positives increases massively due to cross-reaction with other (corona) viruses in the "flu

External peer review of the RTPCR test to detect SARS-CoV-2 reveals 10 major scientific flaws at the molecular and methodological level: consequences for false positive results.https://t.co/mbNY8bdw1p pic.twitter.com/OQBD4grMth
— Dr. Thomas Binder, MD (@Thomas_Binder) November 29, 2020

4/12

Even asymptomatic, previously called healthy, people are tested (en masse) in this way, although there is no epidemiologically relevant asymptomatic transmission. This means that even healthy people are declared as COVID

Thread web\u2b06\ufe0f\u2b07\ufe0f

The fabrication of the "asymptomatic (super) spreader" is the coronation of the total nons(ci)ense in the belief system of #CoronasWitnesses.

Asymptomatic transmission 0.7%; 95% CI 0%-4.9% - could well be 0%!https://t.co/VeZTzxXfvT
— Dr. Thomas Binder, MD (@Thomas_Binder) December 16, 2020

5/12

Deaths within 28 days after a positive RT-PCR test from whatever cause are designated as deaths WITH COVID. This means that other causes of death are reclassified as

8/8

By the way, who the f*** created this obviously (almost) worldwide definition of #CoronaDeath?

This is not only medical malpractice, this is utterly insane!https://t.co/FFsTx4L2mw
— Dr. Thomas Binder, MD (@Thomas_Binder) March 24, 2020