Carp can switch over to an anaerobic metabolism and quietly exhale booze until the situation gets better.
An interesting thing about carp is that they can go into anoxic hibernation and switch to an anaerobic metabolism based on converting glycogen to ethanol.
The waste ethanol is diffused out the gills
Carp can switch over to an anaerobic metabolism and quietly exhale booze until the situation gets better.
In theory, if you spent a few thousand years breeding carp for it, you could use them to make booze.
They'd be enormous, almost entirely glycogen deposits with a fish added as an afterthought.
You, a human, have only about 100 grams of glycogen in your liver, about 400 more grams in your skeletal muscles. Call it 500 grams total.
Carp can be 12% glycogen by weight.
A 75kg human would need to have 9kg of glycogen to match their carp friend.
https://t.co/Bt29kS7pO5
Call it 18 days worth. You'd need a lot more if you wanted to beat your carp friend at holding your breaths.
You currently do this in the liver, by further metabolizing ethanol into acetaldehyde.
You do this completely differently depending on whether you're a fetus or not.
https://t.co/gsnliVwr3G
You're gonna need a bigger liver or a better way
You're gonna scare your carp friends by pissing vinegar at them.
More from Anosognosiogenesis
So I've mentioned the sharpie test and the tueller drill.
Another reason you are dead within 1.5 seconds of encountering your first fast zombie, is adrenaline.
Most people who get attacked with a knife and survive to talk about it, say they never even knew a knife was there.
Or that they'd been stabbed, until after the fact.
In many cases, they think they'd just been punched, and are completely surprised
One reason the adage is "the winner is the one who dies in the ambulance, not the gutter," is because it's entirely possible to receive a fatal wound, not realize it, and then inflict a fatal wound on the other guy without *him* realizing it.
A dozen times within 30 seconds.
The marker drill teaches how you *will* get cut, fatally, without realizing it.
In full adrenaline freakout, this is even more pronounced.
Another reason you are dead within 1.5 seconds of encountering your first fast zombie, is adrenaline.
The Tueller Drill is interesting.https://t.co/D6p3zRRV52
— Anosognosiogenesis (@pookleblinky) December 20, 2020
Most people who get attacked with a knife and survive to talk about it, say they never even knew a knife was there.
Or that they'd been stabbed, until after the fact.
In many cases, they think they'd just been punched, and are completely surprised
One reason the adage is "the winner is the one who dies in the ambulance, not the gutter," is because it's entirely possible to receive a fatal wound, not realize it, and then inflict a fatal wound on the other guy without *him* realizing it.
A dozen times within 30 seconds.
The marker drill teaches how you *will* get cut, fatally, without realizing it.
In full adrenaline freakout, this is even more pronounced.
The US immigration act of 1907 signed by Teddy Roosevelt: ableist as hell. https://t.co/ficeXOImo5
One theory for why the Spanish flu was so unusually lethal for young people:
They hadn't lived through the previous flu pandemic of 1889-1890 (https://t.co/OiDZYtdbWx) that killed about 1 million people. And thus had no carryover immunity.
It's suspected that the 1889 pandemic was not influenza, but a coronavirus.
The 1889 virus spread rapidly, killing mostly the elderly.
The 1889 virus was the first truly modern pandemic: people knew about germs, it spread via trains, it spread at the speed of modern transportation and commerce
Today's covid denialists are tomorrow's openly eugenicist "these disabled people are a drain on society"
— Anosognosiogenesis (@pookleblinky) November 29, 2020
Literally. 13 years after the Spanish flu, the very first people the nazis targeted were disabled people.
What caused a lot of those disabilities, you think?
One theory for why the Spanish flu was so unusually lethal for young people:
They hadn't lived through the previous flu pandemic of 1889-1890 (https://t.co/OiDZYtdbWx) that killed about 1 million people. And thus had no carryover immunity.
It's suspected that the 1889 pandemic was not influenza, but a coronavirus.
The 1889 virus spread rapidly, killing mostly the elderly.
The 1889 virus was the first truly modern pandemic: people knew about germs, it spread via trains, it spread at the speed of modern transportation and commerce
So you want to generate interesting melodies.
1. Make a file called 1235.txt containing, one per line, all 24 unique permutations of the elements 1 2 3 5.
2. Cp 1235.txt to D.txt
3. Use sed to convert the numbers in D.txt to notes. Now you have 24 permutations of the major tetrachord in D.
4. Play them each. If it sounds like it increases tension, mark the beginning of that cell in 1235.txt with a +. If it sounds like it decreases tension, mark with a -.
Now those 24 melodic cells are divided into two groups: tension increasers and resolvers.
5. Rinse and repeat for all 12 keys.
You now have 13 plaintext files, filled with stuff like + 1 2 5 3 and - D E F# A
6. Figuratively roll dice to decide, given a +/- cell, what the next cell should be.
33% chance a + follows a +, etc.
Now you're outputting a stream of dynamic tensions: ++-+++-+-+---+ etc
1. Make a file called 1235.txt containing, one per line, all 24 unique permutations of the elements 1 2 3 5.
Claude Shannon made this machine to play the hex board game.
— Anosognosiogenesis (@pookleblinky) January 21, 2021
It is literally just a mesh of resistors and some light bulbs. No logic gates, no programming, nothing at all resembling AI.
Check it out: https://t.co/Zoyc9TmBcN pic.twitter.com/EANeMosPhT
2. Cp 1235.txt to D.txt
3. Use sed to convert the numbers in D.txt to notes. Now you have 24 permutations of the major tetrachord in D.
4. Play them each. If it sounds like it increases tension, mark the beginning of that cell in 1235.txt with a +. If it sounds like it decreases tension, mark with a -.
Now those 24 melodic cells are divided into two groups: tension increasers and resolvers.
5. Rinse and repeat for all 12 keys.
You now have 13 plaintext files, filled with stuff like + 1 2 5 3 and - D E F# A
6. Figuratively roll dice to decide, given a +/- cell, what the next cell should be.
33% chance a + follows a +, etc.
Now you're outputting a stream of dynamic tensions: ++-+++-+-+---+ etc
More from Science
JUST ONE PERSON—UK 🇬🇧 scientists think one immunocompromised person who cleared virus slowly & only partially wiped out an infection, leaving behind genetically-hardier viruses that rebound & learn how to survive better. That’s likely how #B117 started. 🧵 https://t.co/bMMjM8Hiuz
2) The leading hypothesis is that the new variant evolved within just one person, chronically infected with the virus for so long it was able to evolve into a new, more infectious form.
same thing happened in Boston in another immunocompromised person that was sick for 155 days.
3) What happened in Boston with one 45 year old man who was highly infectious for 155 days straight before he died... is exactly what scientists think happened in Kent, England that gave rise to #B117.
4) Doctors were shocked to find virus has evolved many different forms inside of this one immunocompromised man. 20 new mutations in one virus, akin to the #B117. This is possibly how #B1351 in South Africa 🇿🇦 and #P1 in Brazil 🇧🇷 also evolved.
5) “On its own, the appearance of a new variant in genomic databases doesn’t tell us much. “That’s just one genome amongst thousands every week. It wouldn’t necessarily stick out,” says Oliver Pybus, a professor of evolution and infectious disease at Oxford.
2) The leading hypothesis is that the new variant evolved within just one person, chronically infected with the virus for so long it was able to evolve into a new, more infectious form.
same thing happened in Boston in another immunocompromised person that was sick for 155 days.
3) What happened in Boston with one 45 year old man who was highly infectious for 155 days straight before he died... is exactly what scientists think happened in Kent, England that gave rise to #B117.
Immunocompromised 45 year old suffered from #COVID19 for 155 days before he died. The virus was changing very quickly inside the man's body\u2014it acquired a big cluster of >20 mutations\u2014resembled the same ones seen in #B117 & #B1351. (NPR audio Part 1 of 2)\U0001f9f5https://t.co/7kWiBZ1xGk pic.twitter.com/ZJ7AExB78Y
— Eric Feigl-Ding (@DrEricDing) February 8, 2021
4) Doctors were shocked to find virus has evolved many different forms inside of this one immunocompromised man. 20 new mutations in one virus, akin to the #B117. This is possibly how #B1351 in South Africa 🇿🇦 and #P1 in Brazil 🇧🇷 also evolved.
2) NPR report audio part 2 of 2:
— Eric Feigl-Ding (@DrEricDing) February 8, 2021
Dr. Li couldn't believe what they found. "I was shocked," he says. "When I saw the virus sequences, I knew that we were dealing with something completely different and potentially very important." pic.twitter.com/HT3Yt6djFd
5) “On its own, the appearance of a new variant in genomic databases doesn’t tell us much. “That’s just one genome amongst thousands every week. It wouldn’t necessarily stick out,” says Oliver Pybus, a professor of evolution and infectious disease at Oxford.
