BC LOCKDOWNS
Saved by @trestrange

I’m not just a lockdown sceptic. I know lockdowns increase infections rather than decrease them because I read research first hand. I’m a citizen in a democracy who wants Government policy to be evidence-based, not epidemiological modelling based. Crazy, I know.

 Lockdowns increase infections because they lower immunity & therefore increase the disease. After hospitals & care homes households account for the largest number of transmissions. Schools/universities act as a break in transmission of the disease. All evidence based.
Transmission does not mean infection. I can transmit SARSCoV2 but I can't transmit Covid19. Whether SARSCoV2 develops at all into mild or severe Covid19 depends entirely on the immune system of the recipient. Early treatment prevents severe Covid19, the right treatment cures it.
"Children act more as a brake on infection," said Prof. Reinhard Berner, the head of pediatric medicine at Dresden University Hospital and leader of the study. "Not every infection that reaches them is passed on."
https://t.co/FokkJhgtzM
Closing schools increases transmission of respiratory viral infection because children act as a break on the community transmission of the virus. We have known this since 1918:
https://t.co/TPRYQ1LAAJ
"We confirm that adding school and university closures to case isolation, household quarantine, and social distancing of over 70s would lead to more deaths compared with the equivalent scenario without the closures of schools and universities."
https://t.co/99FR4IqbJj
"Stringency of the measures settled to fight pandemia, including lockdown, did not appear to be linked with death rate."
https://t.co/5N06jgA0Bg
"We explored two MODELS DEVELOPED BY IMPERIAL COLLEGE..Inferences on effects of Non Pharmaceutical Interventions are non-robust and highly sensitive to model specification. CLAIMED BENEFITS OF LOCKDOWN APPEAR GROSSLY EXAGGERATED."
https://t.co/gX6xuQz5EY
There is no correlation whatsoever between mortality rates & lockdowns. There are instead distinct correlations between mortality rates & vitamin D levels:
"Correlations have been shown between the historic prevalence of vitamin D deficiency and COVID-19 mortality per million by country. This has been shown for European countries"
https://t.co/0FwTjxpKtR
There are also noticeable correlations between Omega-3 levels & low mortality rates:
"Global survey of the omega-3 fatty acids, docosahexaenoic acid and eicosapentaenoic acid in the blood stream of healthy adults"
Green=High Omega-3 levels
Red=Low Omega-3 levels
https://t.co/h0HzV1Y5gN
Why vitamin D rather than lockdowns reduces mortality:
"Vitamin D deficiency enhances the cytokine storm, thereby, it is a risk factor for and/or a driver of the excessive and persistent inflammation, which is a main characteristic of ARDS and may be considerably lethal in subjects with SARS‐CoV‐2 infection"
https://t.co/vAw4pphsSC
"When a T cell is exposed to a foreign pathogen, it extends a signalling device or 'antenna' known as a vitamin D receptor, with which it searches for vitamin D,", and if there is an inadequate vitamin D level, "they won't even begin to mobilize." 
https://t.co/RPagpdYkll
Why Omega 3s reduce mortality. Thread: https://t.co/UuZjlc5Bm1
"The most restrictive non‐pharmaceutical interventions (NPIs) for controlling the spread of COVID‐19 are mandatory stay‐at‐home and business closures..we do not find significant benefits on case growth of more restrictive NPIs."
https://t.co/2STxtMHVmA

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Robin Monotti MA FRSA\u2b50
Robin Monotti MA FRSA⭐...
@robinmonotti
"COVID-19 vaccines designed to elicit neutralising antibodies may sensitise vaccine recipients to more severe disease than if they were not vaccinated. Vaccines for SARS, MERS and RSV have never been

"data generated in the development & testing of these vaccines suggest a serious mechanistic concern: that vaccines designed empirically using the traditional approach (consisting of the unmodified or minimally modified coronavirus viral spike to elicit neutralising antibodies)"

"be they composed of protein, viral vector, DNA or RNA and irrespective of delivery method, may worsen COVID-19 disease via antibody-dependent enhancement (ADE)."

"This risk is sufficiently obscured in clinical trial protocols and consent forms for ongoing COVID-19 vaccine trials that adequate patient comprehension of this risk is unlikely to occur, obviating truly informed consent by subjects in these trials."

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Robin Monotti MA FRSA \u2b50
Robin Monotti MA FRSA ...
@robinmonotti
They are now saying my tweets are misleading. It's another lie. They are the ones who are misleading, and I will now prove it in this thread. They are lying about the safety of the vaccines:

"Why was vaccination with the COVID-19 vaccine AstraZeneca suspended?" Paul-Erlich Federal Institute for Vaccines & Biomedicines,

"A specific form of severe cerebral venous thrombosis associated with platelet deficiency (thrombocytopenia) and bleeding has been identified in seven cases (as of 15 March 2021) in temporal association with vaccination with COVID-19 Vaccine AstraZeneca."

"1. It is a very serious disease that is also difficult to treat. Of the seven affected individuals, three individuals had died."

"2. The affected individuals had ages ranging from about 20 to 50 years."
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Robin Monotti Graziade...
@robinmonotti
LIST OF QUESTIONS FOR MPS
From @ClareCraigPath and Dr Jonathan Engler:
.
1.Why are SARS-CoV-2 antibody levels flat or dropping across all age groups since May if the pandemic is still going?

2.What percentage of the population is assumed to have had prior immunity to SARS-CoV-2 in the SAGE forecasting models?

3.Why do 50% of household members not catch SARS-CoV-2 from infected persons with whom they live?

4.Why have Japan and South Korea not had any serious outbreak if the human species has no prior immunity to SARS-CoV-2?

5.What percentage of the population of the UK is assumed to be immune to COVID-19 (including prior immunity) as of this date?
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Robin Monotti
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I have now re-examined this document:


It clearly does indicate both the risks of bacterial infection & to prescribe broad spectrum antibiotics as part of treatment:
"Collect blood cultures for bacteria that cause pneumonia and sepsis, ideally before antimicrobial therapy. DO NOT
delay antimicrobial therapy"

"6. Management of severe COVID-19: treatment of co-infections
Give empiric antimicrobials [broad spectrum antibiotics] to treat all likely pathogens causing SARI and sepsis as soon as possible, within 1 hour
of initial assessment for patients with sepsis."

"Empiric antibiotic treatment should be based on the clinical diagnosis (community-acquired
pneumonia, health care-associated pneumonia [if infection was acquired in health care setting] or sepsis), local epidemiology &
susceptibility data, and national treatment guidelines"

"When there is ongoing local circulation of seasonal influenza, empiric therapy with a neuraminidase inhibitor [anti-viral influenza drugs] should
be considered for the treatment for patients with influenza or at risk for severe disease."
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"PCR is just a process..it does not tell you that you are sick."

Kary Mullis Inventor of the PCR test & Nobel Prize in
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