https://t.co/jBQlWRxcEL
The evidence based science shows that medical face masks for the healthy do not alter rates of community transmission of SARSCoV2 while they contribute to the plastic pollution of planet. Cloth & masks of other materials increase rates of infection through nebulization spread.
https://t.co/jBQlWRxcEL
https://t.co/djT0mfutv9
Prof. Carl Heneghan, Oxford University: "The high quality trial evidence for cloth masks suggest they increase your rate of reinfection."
"Speaking through some masks dispersed largest droplets into a multitude of smaller droplets..smaller particles are airborne longer than large droplets (larger droplets sink faster), a mask might be counterproductive."https://t.co/jBQlWRxcEL pic.twitter.com/XeKYFmjsGs
— Robin Monotti (@robinmonotti) January 15, 2021
https://t.co/BxVILCjtM0
4/In humans, larger aerosols deposit in upper throat, nose, & tracheobronchial region of the lung. Medium-sized aerosols mostly deposit in small airways further down. The really small aerosols <1 \u03bcm can penetrate all the way to the alveoli - the basic units for gas exchange. 4/7 pic.twitter.com/9ZJb4JrZqI
— Dr. Ali Nouri (@AliNouriPhD) December 29, 2020
https://t.co/icOmr0advF
1/\U0001f4ccNIH Study: COVID-19 severity could depend on route of infection: Infection through inhaling #Airborne virus could lead to more severe disease than infection from fomites (contact w/ contaminated objects.) To test it, hamsters were infected via the different routes. 1/7\U0001f9f5 pic.twitter.com/9lX7sMrKUG
— Dr. Ali Nouri (@AliNouriPhD) December 29, 2020
https://t.co/A6YBcGZLVN
2 Smaller particles float in air for longest, larger particles drop
3 Smaller particles get deeper into the lungs, leading to more severe infection & higher mortality
4 Reduce oxygen & increase CO2
https://t.co/77i7BWcpwF
6/Loss of these alveolar cells results in alveolar instability, lung flooding, and respiratory failure - Acute Respiratory Distress Syndrome (ARDS). ARDS causes further damage through a hyperactive immune response of neutrophils and cytokines that damage alveoli even more. 6/7 pic.twitter.com/YicRqalyH5
— Dr. Ali Nouri (@AliNouriPhD) December 29, 2020
https://t.co/gDUiS1Ujet
https://t.co/lLvsmtGUmb
https://t.co/WRFEAEE5wW
People who got infected:
1.8% in the mask group
2% in group who wore masks exactly as instructed
2.1% in the no mask group:
"The difference observed was not statistically significant."
https://t.co/rCtekH6eoT
>>
"Droplets are larger (>5 μm) rapidly drop to the ground by force of gravity.
Aerosols are smaller (≤5 μm) rapidly evaporate, leaving behind droplet nuclei that are small enough & light enough to remain suspended in the air for hours (analogous to pollen)."
7/Viewing transmission through the lens of bioaerosol mechanics highlights the need for mitigation approaches such as ventilation and other measures, and new therapeutic interventions, like surfactant therapy to alleviate ARDS symptoms. 7/7https://t.co/GLjdk9gkZU
— Dr. Ali Nouri (@AliNouriPhD) December 29, 2020
https://t.co/7VSaerYh5p
FACT: The PCR or lateral flow tests do not test for Covid19 nor do they test for even a single or even many SARSCoV2 particles or virions.
— Robin Monotti (@robinmonotti) January 12, 2021
More from Robin Monotti
Here is the Italian-EU scientific study indicating SARSCoV2 replicates in bacteria, not only human cells, and that is why antibiotics work and these vaccines will not:
"The preliminary results suggest that SARS-CoV-2 replicates in bacterial
2 of the 4 authors of the study work at the European Commission. Another works at an Italian medical research facility called Craniomed: https://t.co/EETSM3nb3T
You can find all of CRANIOMED's Carlo Brogna's published scientific research articles here, take a look:
#BMJResearch update: Corticosteroids probably reduce mortality and mechanical ventilation in patients with covid-19 compared with standard care, whereas azithromycin, hydroxychloroquine, interferon-beta, and tocilizumab may not reduce either https://t.co/oQ3lTWUqaz
— The BMJ (@bmj_latest) December 18, 2020
This meta-analysis of controlled trials only looks at hospitalized patients. How long were the patients ill for before being hospitalized? One week? Two? Three? Too late for zinc ionophores (HCQ) (+ZINC? No zinc no point..) to work. Severe illness becomes bacterial in nature.
Was azythromycin administered when the bacterial infections were also too advanced? I have seen Azythromycin work with my very own eyes but that's not to say that if administered too late it may not save the patient. How many patients were given AZT & ventilated? It's all timing.
All the meta-analysis is telling us is if you leave it too late you may have missed the early window for antiviral zinc treatment (Zn+HCQ) & that if you are given AZT when you are ventilated or very severe it may too late for it to save you & corticosteroids may be last resort.
And of course antibiotics need also probiotics, or they may harm the bacterial flora which is part of the immune response. Difficult to tell from a meta-analysis how this problem was managed.
More from All
Viruses and other pathogens are often studied as stand-alone entities, despite that, in nature, they mostly live in multispecies associations called biofilms—both externally and within the host.
https://t.co/FBfXhUrH5d
Microorganisms in biofilms are enclosed by an extracellular matrix that confers protection and improves survival. Previous studies have shown that viruses can secondarily colonize preexisting biofilms, and viral biofilms have also been described.
...we raise the perspective that CoVs can persistently infect bats due to their association with biofilm structures. This phenomenon potentially provides an optimal environment for nonpathogenic & well-adapted viruses to interact with the host, as well as for viral recombination.
Biofilms can also enhance virion viability in extracellular environments, such as on fomites and in aquatic sediments, allowing viral persistence and dissemination.
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The story doesn\u2019t say you were told not to... it says you did so without approval and they tried to obfuscate what you found. Is that true?
— Sarah Frier (@sarahfrier) November 15, 2018
In the spring and summer of 2016, as reported by the Times, activity we traced to GRU was reported to the FBI. This was the standard model of interaction companies used for nation-state attacks against likely US targeted.
In the Spring of 2017, after a deep dive into the Fake News phenomena, the security team wanted to publish an update that covered what we had learned. At this point, we didn’t have any advertising content or the big IRA cluster, but we did know about the GRU model.
This report when through dozens of edits as different equities were represented. I did not have any meetings with Sheryl on the paper, but I can’t speak to whether she was in the loop with my higher-ups.
In the end, the difficult question of attribution was settled by us pointing to the DNI report instead of saying Russia or GRU directly. In my pre-briefs with members of Congress, I made it clear that we believed this action was GRU.
I believe that @ripple_crippler and @looP_rM311_7211 are the same person. I know, nobody believes that. 2/*
Today I want to prove that Mr Pool smile faces mean XRP and price increase. In Ripple_Crippler, previous to Mr Pool existence, smile faces were frequent. They were very similar to the ones Mr Pool posts. The eyes also were usually a couple of "x", in fact, XRP logo. 3/*
The smile XRP-eyed face also appears related to the Moon. XRP going to the Moon. 4/*
And smile XRP-eyed faces also appear related to Egypt. In particular, to the Eye of Horus. https://t.co/i4rRzuQ0gZ 5/*