BC SCIENCE
Saved by @ThomassRichards

In a number of Sunday papers:
Scientific advisers said it was “hard to see” when the restrictions would end as new variants continued to emerge, delivering a potentially devastating blow to the travel industry. The prime minister said that after progress with vaccinations...

 ...”what we don’t want to see is all that work undone by the arrival of a new variant that is vaccine-busting”.
The idea is planted in people’s mind that this virus is mutating in such a way as to evade prior immunity. This is completely unfounded, certainly as regards immunity..
...gained naturally, after repelling the virus (which well around 99.9% of the population do, fewer, obviously in the very elderly & already ill, more in those younger & well). It’s important to appreciate that upon infection, the human immune system cuts up an infectious...
...agent into short pieces. Each of these short pieces of protein are presented to other cells in the immune system, like an identity parade. Our cells have a truly astonishing range of abilities to recognise different protein structures, and there will be some which recognise...
...each of the pieces of the invader. The cells which recognise a piece are instructed to multiply selectively so that, after a few days to a couple of weeks, our bodies contain large numbers of virus piece specific cells. These have a range of functions. Some make antibodies...
& others are programmed to kill cells infected by the virus, recognised by displaying on their surface signals that tell the body that they’ve been invaded. In almost all cases, accompanied or not by symptoms (ranging from mild to severe) this smart adaptive system overcomes...
...the infection. Crucially and against what SAGE & Govt are saying or implying, this event leaves you with many different kinds of long-lived ‘memory’ cells which, if you’re infected again, rapidly wipe out any attempt at reinfection. So you won’t again be made ill by the...
...same virus, and because the virus is simply not permitted to replicate, you are also no longer able to participate in transmission.
To mutations & variants. Many viruses are error-prone when they replicate in your cells. They make “typos” so the virus which results is...
...slightly different from the parent virus. Sometimes that small changes makes no difference to the behaviour of the virus. Other times, the change renders the virus incapable of something important to its survival. It’s possible a change makes it slightly better at surviving...
...and so over time, it becomes a higher proportion of viruses sequenced from clinical samples. The general ‘direction of travel’ is to become less injurious but easier to transmit, eventually joining the other 40 or so viruses which cause what we collectively term ‘the common...
...cold”. What generally doesn’t happen is for mutants to become more lethal to the hosts (us). But the key point I wanted to get across is just how large SARS-COV-2 is. I recall it’s of the order of 30,000 letters of genetic code which, when translated, make around 10,000...
...amino acids in several viral proteins. Now you can see that the kinds of numbers of changes in the letters of the genetic code are truly tiny in comparison with the whole. 30 letter changes might be roughly 0.1% of the virus’s code. In other words, 99.9% of that code is not...
...different from the so-called Wuhan strain. Similarly, the changes in the protein translated from those letter code alterations are overwhelmed by the vast majority of the unchanged protein sequences. So your immune system, recognising as it does perhaps dozen of short pieces..
...will not be fooled by a couple of small changes to a tiny fraction of these. No: your immune system knows immediately that this is an invader it’s seen before, and has no difficulty whatsoever in dealing with it swiftly & without symptoms. So it’s a scientifically invalid...
...reason that’s been given for really extreme responses, closing borders to almost halt international travel. To the ‘vaccines’, they are unusually narrow in the way they educate your immune system, because instead of exposing your body to the whole range of pieces of broken...
...or inactivated virus, a time-honoured method of preparing vaccines, all of these instead present only the spike protein. This still comprises 10 or so distinctly immunologically different pieces, so even if mutations did change a couple of these, the majority of the pieces...
...of the mutated virus will still be unchanged & recognised by the vaccine-immune system or the virus-infected immune system & a prompt, vigorous response will still protect you. The most limited vaccine is, as I understand the molecular biology, the Pfizer / BioNTech version.
This encodes only a part of the spike protein and, if mutations were to evolve away from any of the ‘vaccines’, it would therefore be reasonable to expect it to be this one. But even in the worst case, the other vaccines (& there may yet be more) could be used to protect the...
...most vulnerable people. So what is happening in the name of saving lives simply doesn’t stand up to scientific scrutiny.
Why these incorrect stories are being spun, I’ve no idea, that not being a scientific matter. But spin & completely inappropriate & severe restrictions...
...have been applied, which gradually has the effect of closing down movements. Such movements you’ll appreciate are irrelevant to the natural course of the virus & all its natural progeny through the human population. Note that in their most up to date pandemic preparedness...
...planning documents, WHO specifically excludes closing of borders & lockdowns as ineffective & hugely damaging. So I’ve tweeted my way around the houses in an attempt to explain to you why what is happening is wholly inappropriate & specifically excluded by WHO guidance.
I never encourage anyone to break any rules. I’ve always respected the law. But what I’m afraid I do have to urge you to do is do a little research of your own to test whether what SAGE & Govt is telling you marries up with the pre-2020 scientific literature & official guidance..
...or whether it doesn’t. If you find one occasion where what you’re being told runs directly contrary to pre-2020 science & guidance, congratulations! You’ve discovered that you’re being misled & lied to. As I’ve said, science doesn’t give me any advantages in trying to work...
...out “why?”. But once you’ve realised that what you’re being told is not correct, I hope you’ll have the courage to accept what that means, and not try to build an argument which defends that incorrect argument. In the end, it’s totally up to you. I’m just an ordinary and...
...independent scientist who noticed, very early on, that lots of things we were told weren’t correct & instead we’re actively damaging. Sadly, it’s not improved since in any way. Good luck!

More from Yardley Yeadon

Yardley Yeadon
Yardley Yeadon
@MichaelYeadon3
@scottorandojin What if the false diagnosis rate was 90%?
Don’t misunderstand me. I’m a thorough believer in the existence of this virus.

To exemplify: the NHS is under severe winter pressure. Many staff are unavailable, largely due to forced isolation from testing. Also, I’m told the adaptations to the conditions make normal work almost impossible. Trying to keep apart patients in different categories must be...

...incredibly stressful. I doubt I could ever have coped. Add to this the real stream of sick people & it’s a complete nightmare. My sympathies won’t be accepted but I offer them anyway. As to “the deaths”, do you mean the reported count of “covid19 deaths”? Everyone is doing...

...their very best. But as to that count: it’s nothing to do with clinical judgement. A covid19 death is a death from any cause within 28d of a positive test. So once a person is labelled by a positive test, if they then die, they’re a covid19 death. Back to my substantive...

...question: how reliable is that labelling? It’s not a clinical diagnosis. We’re not used to this idea. Never before in medical history has someone had a disease (let alone cause of death) been defined by the result of a single year alone. Yet that’s where we are. I think that..
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Yardley Yeadon
Yardley Yeadon
@MichaelYeadon3
https://t.co/xvP6tVlIaE
Quite a surprise. Ferguson isn’t qualified to comment on such things. He lacks understanding of basic biological concepts, partly explaining why his predictions are often so extreme.


That said, I’ll take him as an ally. It’s clear we’re already at herd immunity. How can I be sure? London had a huge peak of excess deaths in spring. The timing of peak deaths showed clearly that the pandemic was self-limiting. Recall much debate about whether the interval...

...between lockdown & peak deaths was long enough for lockdown to have been the cause of reducing deaths? The answer was “no”. But it was close & I can understand people thinking “well, it’s close, so perhaps it was lockdown that did it?”, thereby establishing a foundational...

...myth favoured by those who genuinely believe lockdown saves lives (it clearly doesn’t).
I’ll get to London & here immunity. But those wavering about the evidence about lockdown in spring really will benefit from cogitating on this graph.


In blue are the 30 trusts with the earliest date of peak deaths in spring. In red, the 30 with the latest peak deaths. For me, this is proof that national lockdown, which began Mar 23, played no role whatsoever in turning the pandemic. You’ll see some areas had peak daily...
SCIENCE
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Yardley Yeadon
Yardley Yeadon
@MichaelYeadon3
@ukiswitheu I invite people to run the thought experiment: “what if the ‘cases’ data is inaccurate?”
Ignore ‘cases’, look instead only at excess deaths (per M Levitt’s tweet). Does that look characteristic of an epidemic? It’s completely diff from spring or any winter flu outbreak.
London:


Can anyone explain why there is no ‘2nd wave’ of excess deaths in London, without invoking herd immunity?
It’s not lockdown. See NW England:
This is the largest #SecondaryRipple (which I predicted).


https://t.co/b0rT5Lq9HI
Now check the 3 predictions I made months ago. They’ve all happened. Compare predictions from SAGE’s statements: they’re all wrong.
Even neutrals at this point might ask themselves “if he’s been right on all predictions, maybe he’s correct now?”


I’ve been saying since the Lighthouse Labs got up & running that I’m deeply sceptical about the trustworthiness of their ‘cases’ data. I showed how, at low virus prevalence, the PCR mass testing data was throwing out potentially 90% positives being

https://t.co/t4qQN4rH0u
I got ‘fact checked’ a LOT over that statement. This paper just published, about precisely that time period I speculated about. Turns out that high-80s% of Dr Healy’s positives by PCR were FALSE. This alone is sufficient in my view to throw severe doubt...
FOR LATER READ
Yardley Yeadon
Yardley Yeadon
@MichaelYeadon3
@DesmondSwayne Sir Desmond is right. SAGE makes mathematical predictions based on flawed models.
I chose to stick my neck out almost 4mo ago here:
https://t.co/b0rT5Lq9HI
I experienced much anxiety doing this, aware that, if I was badly wrong here, I would be destroyed as a contributor.

That said, I thought the immunology so clear that I made three, specific & testable predictions which should apply if I was right.
Please examine them, on the right, under the pie chart:


In particular, I predicted that the epidemic would not reignite in London. Obviously, we have many “cases” but I urge people to disregard these & instead to look at excess deaths, on the principle that a severe (occasionally lethal) respiratory virus would show itself this way.

Here is a chart showing the trend of deaths in London alone over recent years. A prominent peak of deaths is evident as the virus swept through in spring. Also visible the August heatwave. But there are no excess deaths now.
I submit this is a striking example of herd immunity.


I’m still waiting for SAGE or indeed anyone to provide an alternative explanation for the sharp nature of the spring excess deaths peak which doesn’t involve herd immunity or otherwise removing from the pool those vulnerable to lethal outcomes (same result). Given this sharp...
MATHS
Yardley Yeadon
Yardley Yeadon
@MichaelYeadon3
I urge all followers who have read my criticisms of PCR mass testing in U.K. to carefully read Mr Fordham’s carefully worded letter. Note that the innovation minister in the Lords, Lord Bethel, already admitted that the PCR system doesn’t have the equivalent of an MOT. https://t.co/zXzeDMKCBb


Without this information it’s impossible to interpret any result. If the oFPR is 4%, for example, and if the true prevalence is 0.3% (it’s probably less), then for every 10,000 tests, 400 positives would be false & 30 positives would be genuine. So 93% of positives are false.

As Mr Fordham points out, almost all policies pivot on PCR mass testing. Hancock previously admitted on talkRADIO to Julia Hartley-Brewer in late summer that the FPR was “just under 1%”. That was a flat lie (possibly inadvertent but he’s never corrected the record). The reason...

...we are sure Hancock told a lie is that they have never known the FPR. Those including Hancock who believe that the oFPR can be estimated by inspection of the lowest positivity ever recorded, while logical, is completely wrong. Changes in personnel, throughout, testing...

...architecture & the like can radically alter the oFPR. Since Hancock’s remark in late summer, PCR mass testing has moved into the Lighthouse Labs & this creates a new & urgent need to continually assess oFPR. I’ve good reason to believe it’s now VERY much higher now that the...
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Complex systems are sensitive to original conditions, influenced by feedback loops, and buffeted by random outside variables.
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When respected Scientists resign over the false narrative... Take notice... and thank them.

Google Translated Letter from

Prof. Dr. Thomas Aigner,
Department of Geosciences,
University of Tuebingen, Sigwartstrasse 10,
D-72076 Tuebingen Germany

Dear colleagues,
It was with the greatest astonishment, deepest concern, and indeed bewilderment, that I took note of the "7th ad hoc statement" from the National Academy of Sciences Leopoldina on December 8th, 2020.
In my opinion, this paper is not worthy of an honest, critical-

weighing science oriented towards the service and well-being of man. I do not have any medical expertise. However, as a scientist committed to nothing but the pure truth, I take the liberty of speaking up. I feel very alarmed by several points:

1. On November 27, 2020, a group of 22 internationally recognized experts presented the following report on the PCR test, the linchpin of the "pandemic", for the Eurosurveillance magazine:

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What is carbon capture? And how does it work?

While carbon capture is regularly discussed in the media, no one really ever explains what it is.

Below is a quick thread discussing the technology behind traditional carbon capture 👇

Carbon capture is broadly the "capture" of CO2 emissions from a power plant or other type of industrial facility.

Technology is connected to the "tailpipes" of these facilities and is used to remove CO2 from the plant exhaust.

Once the CO2 is removed from the plant exhaust, it is typically pressurized and sent under ground for permanent storage.

This step is called "sequestration" and is why experts often talks about "carbon capture and sequestration" or "CCS".

So how do you "capture" the CO2 molecules from exhaust?

Currently, one of the most economic forms of carbon capture is called "amine-based" capture.

An "amine" is a special liquid chemical which selectively grabs on to CO2 molecules.

To get the amine to grab on to the CO2, the amine is put into the top of a large column, while the exhaust is put into the bottom of the column.

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Michael Adebayo \U0001f1f3\U0001f1ec\U0001f1eb\U0001f1f7
Michael Adebayo 🇳🇬🇫🇷...
@MichAdebayo
Wonder why your Ph.D./Master's application is being rejected?

Here are some insider reasons (from a committee member) that result in such "Love Letters" 👇

- A Short Thread

1) First, you should know that sometimes, an independent reviewer is required (especially in the US) to review applications even after PIs must have selected their candidates. So, even if any bias slips through the PI, the independent reviewer will pick it up.

2) Having said this, there are three major issues that application reviewers/committees often find with your application. These three components make your applications almost challenging to assess.

3) NUMBER ONE - Incomplete Applications

This occurs when you do not upload the requested documents needed to evaluate your file. Think of a candidate that did not send a GRE score when the institution clearly requires it for admission.

4) NUMBER TWO - Error laden Essays

This is particularly interesting because based on reports from this resource person, it is not particularly about how little your experience is but how you communicate it error-free or at least extensively minimized. This gives headache!
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I took a look at Shell's first ever 1.5C scenario and found that it is... remarkably similar to its “well-below 2C” scenario.

Oil, gas, coal, solar.... all basically unchanged.

The key difference: A new forest the size of Brazil to suck up the extra CO2.


Including "nature-based solutions" in the outlook brings forward the date for net-zero emissions to 2058.

Without them their pathway for CO2 emissions is the same as the previous one.

(It's also towards the higher end of 1.5C emissions pathways.)


The "Brazil-sized" forest idea isn't actually new, it has been kicking around for a couple of years.

It was referenced in the "well-below 2C" scenario although not formally included in it, and Shell's CEO has been framing it as the only viable way of getting to 1.5C.


Fine, but who is going to plant all those trees? Well... Shell says it will plant some of them.

Only yesterday Shell said forests were a key part of its net-zero strategy.

Not everyone is convinced though

https://t.co/RaJm7tOHxb


Given that Shell's 1.5C scenario also sees a big scaling up of bioenergy, the question remains: where are all those trees and bioenergy crops going to go?
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#DoYouKnow -The place where
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There is a Legend #story & it is related to this beautiful #sculpture of Kshetrapuraneswarar & Ambal Lalithambigai #Thread 👇


#Legend #story
It is believed that this is birth place of Kashyapa Maharishi’s sons, Arunan& Garudan. Arunan(charioteer of Surya)was physically handicapped.He was a Shiva devotee.Arunan wanted to visit Kailash to have Shiva Darshan,but Surya Dev denied permission.

So Arunan disguised as a girl & was on his way to Kailash.On the way Surya Dev saw this girl & outraged her modesty.Shiva was very angry with Suryan for misbehaviour & cursed him to get darkened. Whole world plunged into darkness.Surya Dev pleaded to Bhagwan Shiv to forgive him.

Shiva Bhagwan asked him to go to Thirumiyachur & worship him there. As advised, Surya Dev performed rigorous penance, placed murthi of Shiva& Parvathy on an elephant & took them to the clouds.Shiva in this temple is known as Meganathaswamy,Devi is known as Lalithambigai


Even after severe penance,Shiva does appear,so Surya Dev cries out in agony.Disturbed by his call, Parvathy protests.Siva pacifies her & offers relief to Surya Dev.The sculpture of Shiva pacifying Ambal shows her sullen from one angle &smiling from another.
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Low-risk, High-return stocks are what we look for relentlessly

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Here are 10 low-risk, high return stocks (that we ignore):

What I mean by “high-return”?

These stocks have OUTPERFORMED the market:

💹Chart with upwards trend Since IPO
💹Chart with upwards trend Over the last 5 Years or since IPO

These are all long-term winners that will keep on winning

What do We Mean by “low-risk”?

A combination of:

❣️Great management
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❣️Consistent Organic Growth
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❣️Low debt

1-#CDSL

A depository is a big business and will grow continuously, Its a sustainable business
cause "Market hai toh CDSL bhi hai or rahega"


2- #Dixon

High Management Efficiency with a high ROE of 19.30%
Healthy long-term growth as Net Sales has grown by an annual rate of more than 30%
High Institutional Holdings
LOW RISK HIGH RETURN STOCKS , LOW RISK HIGH RETURN STOCKS FOR LONG TERM , LOW RISK STOCK IDEAS