Normally I enjoy the high standards of journalism in @guardian . Not today as disappointed with misleading headline that suggest infections are spreading fastest in children. It'll worry parents/teachers & I doubt most readers will unpick the

The latest REACT1 report shows prevalence of infection in ALL age groups has fallen, including children aged 5-12 from 1.59% in Round 8 to 0.86% in Round 9a. The authors of REACT1 report also (wisely) didn't try to interpret the prevalence figures.
2/
https://t.co/SR9PioFNHd
If this were a research trial you wouldn't place much weight on the age differences in % prevalence because of the wide confidence intervals, i.e. differences weren't statistically significant.
3/
I've previously tweeted on the challenges (& dangers) of interpreting surveillance data. One would need lots more contextual info to make sense of it & arrive at sound conclusions.
4/
https://t.co/CRW6uFXUdf
Undoubtedly some will extrapolate from the prevalence of infection figures in children to other settings i.e. schools based on the headline. I'd advise caution as there is a real risk of over-interpretation through extrapolation of limited data. Association is not causation.
5/
What REACT1 can’t tell us is how infectious children are or how much child infections drive the pandemic. Finding viral RNA on PCR from a child tells you nothing about how much virus they are shedding. Neither can it tell you the direction of transmisison (who infected who).
6/
Those of us in public health are well familiar with Bradford Hill criteria for causation that outlines key factors to consider. It would make sense to apply this approach to this issue.
7/
Is there CONSISTENCY? What’s been reported has been mixed, but @ECDC_EU , @CDCgov , @RCPCHtweets & other expert review groups have concluded the balance of evidence suggests kids aren't key transmitters of infection.
https://t.co/llVkHkwrRS
https://t.co/c8oOgua9FE
8/
Is the SPECIFICITY of the association or are there other confounders? Indeed the REACT1 report clearly show there are multiple other drivers such as deprivation & household size, etc...

9/
What about TEMPORAL SEQUENCE, i.e. does a rise in school outbreaks then follow a rise in community outbreaks? We aren’t seeing that.
https://t.co/VPbAmsbApG
10/
For me what also matters is PLAUSIBILITY & COHERENCE with what is known about the natural history and biology of the disease. Studies have shown that kids, being smaller, produce far fewer aerosols than adults. They also shed less virus than others.
https://t.co/Rv1e2VfJi6
11/
And also

https://t.co/GHa0FoJ34m
https://t.co/vW1b7j9p9j

And we know symptomatic persons shed more virus. So it follows that an adult is more likely to infect a child than the other way.
https://t.co/NpVoTakbzs

12/
This isn't to say that infections from children to adults don’t happen. Of course they do, but at much lower levels than the other way around. We mustn't lose sight of the fact that adults still account for the bulk of infections.

13/
We know that of all settings, household transmission is the highest risk. If infection is introduced into the household the majority of household members are likely to get infected. Community transmission matters.
https://t.co/OvWN28MZ0Y
https://t.co/fEOOvn6eE0

14/
This in no way refutes need for public health measures in schools to reduce risks of infection. Yes precautionary principle is important but need to balance both benefits & harms. Measures must be proportionate to actual (rather than perceived) risk.
https://t.co/VkugKMnS9R

15/
See also @apsmunro's excellent thread that outlines the evidence base.

https://t.co/VkugKMnS9R

More from Education

Our preprint on the impact of reopening schools on reproduction number in England is now available online: https://t.co/CpfUGzAJ2S. With @Jarvis_Stats @amyg225 @kerrylmwong @KevinvZandvoort @sbfnk + John Edmunds. NOT YET PEER REVIEWED. 1/


We used contact survey data collected by CoMix (
https://t.co/ezbCIOgRa1) to quantify differences in contact patterns during November (Schools open) and January (Schools closed) 'Lockdown periods'. NOT YET PEER REVIEWED 2/

We combined this analysis with estimates of susceptibility and infectiousness of children relative to adults from literature. We also inferred relative susceptibility by fitting R estimates from CoMix to EpiForecasts estimates(https://t.co/6lUM2wK0bn). NOT YET PEER REVIEWED 3/


We estimated that reopening all schools would increase R by between 20% to 90% whereas reopening primary or secondary schools alone would increase R by 10% to 40%, depending on the infectiousness/susceptibility profile we used. NOT YET PEER REVIEWED 4/


Assuming a current R of 0.8 (in line with Govt. estimates: https://t.co/ZZhCe79zC4). Reopening all schools would increase R to between 1.0 and 1.5 and reopening either primary or secondary schools would increase R to between 0.9 and 1.2. NOT YET PEER REVIEWED 5/
Working on a newsletter edition about deliberate practice.

Deliberate practice is crucial if you want to reach expert level in any skill, but what is it, and how can it help you learn more precisely?

A thread based on @augustbradley's conversation with the late Anders Ericsson.

You can find my complete notes from the conversation in my public Roam graph:
https://t.co/Z5bXHsg3oc

The entire conversation is on

The 10,000-hour 'rule' was based on Ericsson's research, but simple practice is not enough for mastery.

We need teachers and coaches to give us feedback on how we're doing to adjust our actions effectively. Technology can help us by providing short feedback loops.

There's purposeful and deliberate practice.

In purposeful practice, you gain breakthroughs by trying out different techniques you find on your own.

In deliberate practice, an expert tells you what to improve on and how to do it, and then you do that (while getting feedback).

It's possible to come to powerful techniques through purposeful practice, but it's always a gamble.

Deliberate practice is possible with a map of the domain and a recommended way to move through it. This makes success more likely.

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I just finished Eric Adler's The Battle of the Classics, and wanted to say something about Joel Christiansen's review linked below. I am not sure what motivates the review (I speculate a bit below), but it gives a very misleading impression of the book. 1/x


The meat of the criticism is that the history Adler gives is insufficiently critical. Adler describes a few figures who had a great influence on how the modern US university was formed. It's certainly critical: it focuses on the social Darwinism of these figures. 2/x

Other insinuations and suggestions in the review seem wildly off the mark, distorted, or inappropriate-- for example, that the book is clickbaity (it is scholarly) or conservative (hardly) or connected to the events at the Capitol (give me a break). 3/x

The core question: in what sense is classics inherently racist? Classics is old. On Adler's account, it begins in ancient Rome and is revived in the Renaissance. Slavery (Christiansen's primary concern) is also very old. Let's say classics is an education for slaveowners. 4/x

It's worth remembering that literacy itself is elite throughout most of this history. Literacy is, then, also the education of slaveowners. We can honor oral and musical traditions without denying that literacy is, generally, good. 5/x