BC SCIENCE
Saved by @WebbDarrrel

Today, we are bringing other exciting results involving black holes and AI. We released a new paper:

"Black Hole Weather Forecasting with Deep Learning: A Pilot Study"

Work by Roberta Duarte (@import_robs), Rodrigo Nemmen (@nemmen) and João Paulo Navarro (from @NVIDIABrasil).

 The authors used deep learning to simulate the dynamics of gas accreting onto a black hole, i.e., black hole weather forecasting.

They trained the model (U-Net) with frames from numerical solutions of the hydrodynamical equations.
Numerical simulations are time-consuming. A simple simulation can take as long as 7 days to finish. If we go with more complex simulations, this time may increase.

We want to investigate if deep learning can be a new method to simulate accurately in less time!
In the paper, they discussed two examples:

1- The model simulating only one system after learning only from this system
2- The model simulating an unseen system after training with several systems with different initial conditions
In the first example, they trained the model with a single system and analyzed how the model simulates by iterative predictions.

The result is that the model can simulate up to 8e4 gravitational time accurately with a speed-up of 30000x faster!
In the 2nd example, they fed the model seven different simulations with the same physics but other initial conditions. They informed the model how the initial conditions differ from one to another.

However, they hid one system to understand the generalization power of the model!
They analyzed how the model can simulate an unseen system only by looking at previous systems!

It simulated the unseen system for 4e4 gravitational time, showing that the model can generalize the black hole physics presented in the dataset!
In the second example, the model can also simulate the systems it learned from:
For more details, please check it out on arXiv: https://t.co/VBh3RQnhov

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Michael Lin, PhD-MD
Michael Lin, PhD-MD
@michaelzlin
Fellow academics, when are we going to start fixing our urgent structural problems that strain family bonds and present ongoing barriers to diversifying our educational and scientific leadership? You wonder, what the heck is this guy talking about?
1/n

We’re all very familiar what I am talking about, actually: the impossibility for most academics in the life sciences to do their jobs in a reaonable amount of time, due to more and more demands on writing grants, and to a lesser degree, papers.
2/n

As of yesterday I hadn’t intended to write about this subject, but was wondering as I finished a Saturday of straight work, what happened to all my time? Why has WFH not reduced my pile of tasks to do? Instead why is my to-do list growing bigger and bigger?
3/n

I decided to figure out how much time our jobs actually require. I estimated per year (m=month, w=week, d=day, h=hour):
Teaching: 20h/w x 20w = 400h
Meetings of all sorts: 10h/w x 48w= 480h
Replying to email: 2h/d x 365d = 730h
Attending and giving talks: 2h/w x 50w = 100h
4/n

Papers: If each person needs 2 papers during a 5y stint, and you run a 7.5-person lab, then you need 7.5 papers per 2.5y = 3 papers/y. Throw in a review and it’s 4 papers. If you need 15d at 8h/d per paper, that’s 480h writing papers.
5/n
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Dr. Angela Rasmussen
Dr. Angela Rasmussen
@angie_rasmussen
Hard agree. And if this is useful, let me share something that often gets omitted (not by @kakape).

Variants always emerge, & are not good or bad, but expected. The challenge is figuring out which variants are bad, and that can't be done with sequence alone.


You can't just look at a sequence and say, "Aha! A mutation in spike. This must be more transmissible or can evade antibody neutralization." Sure, we can use computational models to try and predict the functional consequence of a given mutation, but models are often wrong.

The virus acquires mutations randomly every time it replicates. Many mutations don't change the virus at all. Others may change it in a way that have no consequences for human transmission or disease. But you can't tell just looking at sequence alone.

In order to determine the functional impact of a mutation, you need to actually do experiments. You can look at some effects in cell culture, but to address questions relating to transmission or disease, you have to use animal models.

The reason people were concerned initially about B.1.1.7 is because of epidemiological evidence showing that it rapidly became dominant in one area. More rapidly that could be explained unless it had some kind of advantage that allowed it to outcompete other circulating variants.
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Prof Francois Balloux
Prof Francois Balloux
@BallouxFrancois
@mugecevik is an excellent scientist and a responsible professional. She likely read the paper more carefully than most. She grasped some of its strengths and weaknesses that are not apparent from a cursory glance. Below, I will mention a few points some may have missed.
1/


The paper does NOT evaluate the effect of school closures. Instead it conflates all ‘educational settings' into a single category, which includes universities.
2/

The paper primarily evaluates data from March and April 2020. The article is not particularly clear about this limitation, but the information can be found in the hefty supplementary material.
3/


The authors applied four different regression methods (some fancier than others) to the same data. The outcomes of the different regression models are correlated (enough to reach statistical significance), but they vary a lot. (heat map on the right below).
4/


The effect of individual interventions is extremely difficult to disentangle as the authors stress themselves. There is a very large number of interventions considered and the model was run on 49 countries and 26 US States (and not >200 countries).
5/
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agamshastra
agamshastra
@truejainology
Some 1300 years ago in Karnataka there lived a genius who wrote "Gaṇitasārasan̄graha" (which is earliest text devoted entirely to mathematics)

The man who's work later borrowed by Arabs & Europeans

Thread on "Mahāvīracharya" astounding but forgotten legacy


Mahāvirachārya was patronised by rashtrakuta king amoghavarasha

His book"Gaṇita-sāra-saṅgraha"is earliest text devoted entirely to Mathematics

He has described the currently used method of calculating(LCM)of given numbers.The same method was used in Europe later in1500CE

Mahavira wrote "Gaṇita-sāra-saṅgraha" (GSS) in 8th century CE which contains some 1100 slokās & contains elementary topics in arithmetic , algebra , geometry , measurements , logic , number theory , dynamical systems etc

A sheer mathematical genius


He was devout jain
His magnum opus "Ganit sara sangraha " start with a sloka bowing to the glory of the jinas

"संख्याज्ञानप्रदिपेन जैनेंद्रन महत्वेशा,
प्रकाशितम जगतसर्व येन तम प्रणमाम्यहम"

He also praised his predecessor aryabhata , brahmagupta & other greats in his works


The importance & popularity of "Gaṇita-sāra-saṅgraha"was so huge that it enjoyed statues of most important textbook of mathematics in South Indian for over 3 centuries

A large no of manuscript copies of
"Gaṇita-sāra-saṅgraha"whr discovered in Kerala shows it's popularity
SCIENCE
Yardley Yeadon
Yardley Yeadon
@MichaelYeadon3
In a number of Sunday papers:
Scientific advisers said it was “hard to see” when the restrictions would end as new variants continued to emerge, delivering a potentially devastating blow to the travel industry. The prime minister said that after progress with vaccinations...

...”what we don’t want to see is all that work undone by the arrival of a new variant that is vaccine-busting”.
The idea is planted in people’s mind that this virus is mutating in such a way as to evade prior immunity. This is completely unfounded, certainly as regards immunity..

...gained naturally, after repelling the virus (which well around 99.9% of the population do, fewer, obviously in the very elderly & already ill, more in those younger & well). It’s important to appreciate that upon infection, the human immune system cuts up an infectious...

...agent into short pieces. Each of these short pieces of protein are presented to other cells in the immune system, like an identity parade. Our cells have a truly astonishing range of abilities to recognise different protein structures, and there will be some which recognise...

...each of the pieces of the invader. The cells which recognise a piece are instructed to multiply selectively so that, after a few days to a couple of weeks, our bodies contain large numbers of virus piece specific cells. These have a range of functions. Some make antibodies...
SCIENCE

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Mitesh Patel
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Again sharing bank nifty intraday weekly straddle. How to play by selling straddle.
Mostly will work on Monday to Wednesday. Sometimes on Friday.
Why to pay your money to learn this if u can get it here.
U need only practice.
Let’s go.

Say bank nifty future is at 32000.
Sell both 32000 call and put.
Considering current volatile market lest say premium on Monday morning is 350 on both sides.
Sold with 700 points combine premium.
If bank nifty will trade in narrow range say +\- 100 hold it.
If moving 200 points

then exit previous sold straddle. Sell new one. Let’s say BN moved +200
Now sell 32200. If bank nifty will move here within range then u will get profit through theta decay. Follow same procedure for next 150-200 pts move.

This is only for single straddle sell position.

Now in second case sell three straddle. 31900/32000/32100
In case of 100 points move no need to change.
If 200 + then future is 32200.
Exit 31900 and sell 31200 straddle.
If moving another 100 then exit 32000 and sell 32300. Continue same with 100 points move.
If no move then

it will consolidate between 32000-32200 or 32100 to 32300 and u will get theta decay.

This is for normal move. Sudden big move may come anytime. So keep SL in system.
If u r selling at 350-350 then keep SL in system 475 for both legs.
Practice it. No need to attend webinar.
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Nikita Poojary
Nikita Poojary
@niki_poojary
Mr. Vijay Kedia built a net worth of over Rs. 800 crores.

But you probably never read past that headline.

So I binged hours of interviews to learn his investing principles and strategies.

Here are 20 lessons on investing from @VijayKedia1 Sir:

Collaborated with @AdityaTodmal


Before I start, a quick disclaimer, please don't ape his portfolio.

As the entry price, horizon and risk appetite would be far different than yours.

Rather lets draw inspiration and try to learn his stock picking skills.

Now let's DIVE IN👇

1/ Started off as a trader:

- Soon, beginner’s luck started to fade out and loses started to mount, to an extent where his mother was about to sell her ornaments.

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2/ Punjab Tractors:

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Anshul Pandey
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IMPORTANCE, ADVANTAGES AND CHARACTERISTICS OF BHAGWAT PURAN

It was Ved Vyas who edited the eighteen thousand shlokas of Bhagwat. This book destroys all your sins. It has twelve parts which are like kalpvraksh.

In the first skandh, the importance of Vedvyas


and characters of Pandavas are described by the dialogues between Suutji and Shaunakji. Then there is the story of Parikshit.
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It also discusses the evolution of universe.( https://t.co/2aK1AZSC79 )

Next is the portrayal of Vidur and his dialogue with Maitreyji. Then there is a mention of Creation of universe by Brahma and the preachings of Sankhya by Kapil Muni.


In the next section we find the portrayal of Sati, Dhruv, Pruthu, and the story of ancient King, Bahirshi.
In the next section we find the character of King Priyavrat and his sons, different types of loks in this universe, and description of Narak. ( https://t.co/gmDTkLktKS )


In the sixth part we find the portrayal of Ajaamil ( https://t.co/LdVSSNspa2 ), Daksh and the birth of Marudgans( https://t.co/tecNidVckj )

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RISHI ASHTAVAKRA(DEVAL)

Those who are well versed in our ancient scriptures must definitely have come across the name of Ashtavakra Muni. Who was he and what is the story behind his body getting crooked from all sides.

Muni Asit was the son of Pracheta Rishi.


He was childless and this caused a lot of worry to him. But an Akashvani assured him that he should go to Shivji to get all his
worries nullified.

So he along with his wife go to Shivji and after paying him obeisance start singing Shivji's praises.

He describes Shiv as the one who is beyond any other thing, whose glory has no words to describe. After this the couple stands with folded hands in front of Rudradev. The strotras sung in praise of Shiv possess magical qualities even today.

Shiv gets satisfied by them soon and is ready to grant a boon.

Shiva told him that, he very well knew what the Muni desired. He then gives him the "Sansaar Vijay' mantra and asks him to keep reciting it. The Isht devi would come at an appropriate time and fulfill his wish.

Asit Muni kept reciting this mantra for hundred years. The Devi appeared and granted him the boon of a son as a Shivansh. A son was born and was named as Deval. He grew upto be a great scholar well versed and a handsome young man. Asit Muni got him married to Ratnamalavati.
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Aditya Todmal
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